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It is likely an adaptive stress response induced by a disturbance of homeostasis caused by low levels of biotic or abiotic stimuli (Vargas-Hernandez et al., 2017)

GH also activates the MAPK/ERK pathway through Shc recruitment, the PI3K/AKT/mTOR pathway through IRS adaptors, and Src family kinases through a JAK2-independent branch

This is especially important for people with Hashimoto's thyroiditis, the autoimmune thyroid condition where the immune system attacks the thyroid

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4-OH-E 2 is capable of binding ER with a reduced dissociation rate and prolonged activation, thereby inducing cellular growth and proliferation, [6] adenohypophyseal hormone secretion, and prostaglandin production