slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Development of a mouse model expressing a bifunctional glutathione-synthesizing enzyme to study limitation vivo Glutathione-driven redox decisions in cell

Name: slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Development of a mouse model expressing a bifunctional glutathione-synthesizing enzyme to study limitation vivo Glutathione-driven redox decisions in cell
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Glutathione driven redox decisions in cell survival and death Combinatorial GxGxE CRISPR screen identifies SLC25A39 in mitochondrial glutathione transport linking iron homeostasis to OXPHOS Nature Communications SLC25A39 Wikipedia Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis ScienceDirect Controlling glutathione entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease Signal Transduction and Targeted Therapy

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28 When cellular iron is sufficient, transferrin-bound iron is reduced to limit excess iron accumulation

Glutathione (GSH) as distributed by Peptides Lab UK is not intended for, and must not be used for, human consumption, medical treatment, self-administration, veterinary applications, or any use outside of a controlled laboratory environment