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Macromolecular damage via oxidative and/or nitrosative stress, if unchecked, can theoretically contribute to disease development, and an increasing amount of evidence suggests that oxidative and/or nitrosative stress is linked to the pathophysiologic mechanisms of a myriad of human diseases [3,6,4850]

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Excessive autophagy can promote ferroptosis by increasing iron ion content and lipid peroxide accumulation [86]

Hoffman, Gamze Grsoy, Garrett Gee, Gaurang Mehta, Gianfilippo Cop-pola, Gina Giase, Goran Sedmak, Graham D