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The energy deficit causes structural and functional damage to macromolecules by increased ROS that ultimately leads to severe axonal damage

10.1159/000509733 Iorio R, Celenza G, Petricca S: Multi-Target Effects of -Caryophyllene and Carnosic Acid at the Crossroads of Mitochondrial Dysfunction and Neurodegeneration: From Oxidative Stress to Microglia-Mediated Neuroinflammation

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These agents can inhibit the electron transport chain, increasing dopamine turnover and generating harmful reactive oxygen species, causing neuronal damage

Reviewing the potential of AA derivatives to escape from ruminal degradation, Loerch and Oke (1989) reported three main products that promote an increase in Met absorption post-ruminally, known as N-stearoyl-DL-Met, N-oleoyl-DL-Met, and Capryl- caprylic-Met