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Although FOXO4s specific effects on mitochondria are as direct as the other peptides above, it is known to regulate the expression of genes involved in oxidative stress response and energy metabolism, which can impact mitochondrial function indirectly (Liu, 2019)

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However, the pathological mechanisms underlying cerebral ischemia reperfusion injury (CIRI) after CPR are very complex and involve calcium overload, mitochondrial dysfunction, inflammatory responses, apoptosis, the accumulation of oxygen free radicals, and the excessive release of excitatory amino acids[19]

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