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Hemoglobin, heme and hemin produced by old red blood cells trigger ferroptosis in macrophages, monocytes and platelets, thereby regulating immune response

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Collectively, lactate-mediated reprogramming ensures that these long-lived neutrophils actively support tumor progression rather than antitumor immunity 65,66

Nevertheless, evidence suggests that in TAMs and MDSCs, oxidative stress can activate IB kinase (IKK) by promoting ROS production, leading to the degradation of IB protein, the release of NF-B, upregulation of PD-L1, and ultimately reduced anti-tumor T cell activity (194, 195)

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