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Liu T, Peng YF, Jia C, Yang BH, Tao X, Li J

[DOI] [PMC free article] [PubMed] [Google Scholar] 95.Shahverdi A, Moghaddam K, Yazdi M, and Iranshahi M

The function of NPAC/GLYR1 was initially characterized by its ability to bind to the trimethylated histone H3-K36 (H3K36me3) as an epigenetic factor (Fig

A study published in Circulation noted that while these reductions in muscle volume appear proportionate to the weight loss achieved, the concern remains that excessive lean mass loss could lead to sarcopenia, reduced metabolic rate, and poorer long-term outcomes