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Abstract Although oxidative stress has been implicated in the pathogenesis of hypertension in spontaneously hypertensive rats (SHRs), there is little information on the levels of primary antioxidant enzymes status (AOEs) in pre-hypertensive SHR

It also suppressed pro-inflammatory cytokines (e.g., TNF-, IL-6, IL-8, interferon (IFN)-, IL-12p40, IL-1 and IL-17) and enhanced apoptosis-regulating proteins (caspase-8, Bax), highlighting its potential for managing intestinal inflammation

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The subsequent sections describe four key pathophysiological components of asthma: persistent airflow limitation, airway hyperresponsiveness, airway inflammation, and hyperproduction of airway mucus

Sanchez, S., Ricca, R., Joyner, B