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doi: 10.1155/2013/159649

Their simultaneous activation does not produce simple pathway redundancy but rather engages complementary aspects of the tissue repair response, providing the biological rationale for studying whether combination produces additive or synergistic repair outcomes exceeding either peptide alone

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Research has investigated its interactions with nitric oxide (NO) pathways, growth factor modulation (including VEGF and EGF), and the FAK-paxillin signaling cascade involved in tissue remodeling studies

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