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The role of autophagic pathways over the prevention of neurodegeneration has been evidenced by generating selective neural animal models, such as one in which cell-specific Atg5 deletion is controlled, as Atg5 is essential for autophagosome formation and thus, mice deficient for this protein develop progressive deficits in motor function that are accompanied with the accumulation of cytoplasmic inclusion bodies in neurons (Hara et al., 2006)

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and Petrou, Steven (2018)

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FOXO4-DRI Peptide and Cardiovascular Function Research from 2002 (7) has suggested that levels of proteasome enzymes decrease over time