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Mechanistically, BDH1 reduced H3K9 -hydroxybutyrylation at the LCN2 promoter, suppressed LCN2 transcription and NF-B activation, while treatment with the -hydroxybutyrylation inhibitor A485 similarly alleviated the cardiac damage [134]

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Notably, a negative correlation was observed between tumor SLC7A11 expression and T-cell infiltration, while SLC7A11 expression was positively associated with immunosuppressive Th2 infiltrates (Fig