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However, it has been shown that mortality is significantly decreased by the administration of NAC even up to 36 h after the toxic ingestion and that this cohort of patients is less likely to progress to grade III/IV hepatic coma after receiving treatment with NAC[69]

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In conclusion, our findings highlighted that PPARa promoted TSCI recovery by suppressing lipid peroxidation-induced neuronal ferroptosis via the FSP1 pathway, and positioned the PPARa-FSP1 axis as a central mechanism in post-TSCI ferroptosis and a promising therapeutic target

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Keywords: glutathione peroxidase 1, oxidative stress, selenium, selenocysteine, endothelial dysfunction, cardiac protection, angiogenesis, inflammation, atherosclerosis, stroke, neurodegeneration, cancer Graphical Abstract Glutathione peroxidase 1 (GPx1) is a member of a family of antioxidant enzymes that reduce hydrogen peroxide and soluble lipid hydroperoxides using glutathione (GSH) as a source of reducing equivalents 1