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EEC-derived EXOs supplied miR-30c, which blocked BCL9 transcription and suppressed the Wnt/-catenin system, reducing tumor-like characteristics of ectopic ECs in EMS (Zhang et al., 2022a)

3), whose trans isomer degraded the target in K562 cells, with visible light-induced cis isomerization reversing the effect and enabling a reversible knockdown cycle in a tumor model

7 AlexanderL.GriersonD

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